Delayed Polyneuropathy Induced by Organophosphate Poisoning
نویسندگان
چکیده
منابع مشابه
Delayed Polyneuropathy Induced by Organophosphate Poisoning
An 89-year-old man attempted suicide by ingesting a pesticide (trichlorfon). After surviving the initial critical period in the intensive care unit, he developed rapidly progressive distal weakness and sensory disturbance. Electrophysiological examinations revealed sensory motor axonal polyneuropathy. Delayed polyneuropathy is a rare manifestation of organophosphate poisoning. Nerve conduction ...
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Organophosphate compounds are the organic derivatives of Phosphorous containing acids and their effect on neuromuscular junction and Autonomic Synapses is clinically important. After exposure these agents cause acute and sub acute manifestations depending on the type and severity of the agents like Acute Cholinergic Manifestations, Intermediate Syndrome with Nicotinic features and Delayed Centr...
متن کاملClinical characteristics of organophosphate-induced delayed polyneuropathy.
made in our patient because of the presence of highly characteristic melanin deposits in the MRI study. This is the only method to diagnose CNS melanosis in a living patient. Secondly, in our patient there were no clinical indicators suggestive of a developing cutaneous melanoma, viz. changes in colour, size, shape, rapid growth rate, proliferative nodules or ulceration, in any of the GCMN. Hen...
متن کاملDelayed chronic polyneuropathy following organophosphate poisoning: a case report.
Organophosphate induced delayed polyneuropathy (OPIDP) generally first becomes apparent one to three weeks after the acute poisoning and after a more uncertain interval following chronic exposure. The first symptoms are usually calf pain and paresthesia in the feet and hands to be foil owed by weakness, flaccidity, absent tendon reflexes and sometimes sensory loss usually appearing earlier in t...
متن کاملAcute pancreatitis induced by acute organophosphate poisoning.
A 32-year-old male with acute organophosphate poisoning developed hyperglycaemia, glycosuria and ketonuria soon after admission to hospital. Serum amylase estimations suggested a diagnosis of acute pancreatitis. He required insulin therapy to control his hyperglycaemia and the organophosphate poisoning was successfully managed by artificial ventilation, an infusion of pralidoxime and intermitte...
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ژورنال
عنوان ژورنال: Internal Medicine
سال: 2017
ISSN: 0918-2918,1349-7235
DOI: 10.2169/internalmedicine.56.7921